MGH/McLean Residency Training Program in Psychiatry

Massachusetts General Hospital and McLean Hospital, Harvard Medical School

Edition Massachusetts General Hospital, 1999

PART 3 / CH 58

Extrapyramidal Symptoms (EPS)

Amy Gagliardi

A. Overview

Neuroleptic blockade of the D2 receptors in the basal ganglia may cause a variety of unpleasant, frightening and sometimes life-threatening side effects commonly referred to as EPS. While more common with typical neuroleptics, they may also occur with newer agents.

B. General approach

1. Go see the patient: identify the onset, time course and symptoms.

2. Do a targeted physical exam with a focus on the motor system.

3. Perform a brief mental status exam.

4. Treat the symptoms quickly.

C. Acute Dystonia

· Characterized by brief or prolonged muscle contractures, usually of the head, neck, and tongue, resulting in abnormal movements.

· Laryngeal or pharyngeal dystonias, which present with hoarse voice or a choking sensation, are a medical emergency.

· Other presentations include oculogyric crisis (one or both eyes turned upward), tongue protrusion, and torticollis.

· Prevalence = 10%

D. Parkinsonism

· The same triad as idiopathic parkinsonism:

1. resting tremor (rhythmic, 3-6 cycles/second)

2. rigidity (lead pipe continuous or cogwheel)

3. bradykinesia (masklike facies, difficulty initiating movement,

shuffling gait with propulsion and retropulsion, decreased arm

swing while walking, decreased spontaneous movements).

· If there is rigidity and disorientation, this is likely not parkinsonism and NMS should be ruled out.

· Prevalence = 50%

E. Akathisia

· An objective or subjective feeling of restlessness, characterized by pacing, rocking, or rapid alteration of sitting and standing.

· Patients often describe a sense of "I can't stay still" or "I'm jumping out of my skin."

· Often misdiagnosed as anxiety, agitation, psychosis

· Prevalence ³10% of all patients on neuroleptics

F. Extrapyramidal symptoms: diagnosis

onsetrisk factorsddx
acute dystoniarapid - often hours after 1st dose up to 5 days outmale

under 40

high potency meds

seizures

tetanus

parkinsonism2-6 weeks or immed. after dose increasefemale

elderly

previous hx of parkinsonism

other neurologic

illness

Parkinson’s

disease

catatonia

negative sxs of schizophrenia psychomotor retardation

tardive dyskinesia

akathisia1-4 weeks or immed. after dose increasefemale

middle-aged

almost anything:

anxiety, agitation, psychosis, "acting out"

G. Extrapyramidal symptoms: treatment

symptomtreatment recommendations
acute dystoniaIf laryngeal dystonia with airway compromise,

cogentin 4 mg IV/IM

then ativan 2 mg IV/IM slowly if needed

otherwise,

1) cogentin 2 mg IM/IV or

2) benadryl 50 mg IM/IV

If no response in 20 minutes, repeat above.

If no response after two trials, ativan 1 mg IM/IV.

After treatment, neuroleptic can be continued with standing dose of anticholinergic coadministered for two weeks.

If symptoms return, change to lower potency neuroleptic.

parkinsonism1) dose antipsychotic as low as possible, or switch to low potency antipsychotic, and

2) cogentin 0.5-2 mg po bid and/or

3) benadryl 25-50 mg po bid and/or

4) amantadine 100 mg po bid or tid

note: anticholinergics should be stopped after 14 days if asymptomatic because long-term use can increase the risk of tardive dyskinesia

akathisiaIf no other EPS:

1st choice: propanolol 10 to 30 mg po tid

2nd choice: lorazepam 1 mg po tid

3rd choice: cogentin 1 mg po bid

If other EPS present:

1st choice: cogentin 2 mg po bid

2nd choice: cogentin with propanolol as above

3rd choice: cogentin with lorazepam as above

After treatment, neuroleptic may be continued with benzo or beta-blocker indefinitely; anticholinergics should be weaned after two weeks if possible.

source: Hyman SE, Tesar GE (1994). Manual of Psychiatric Emergencies. Little, Brown.